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Table 1 Clinicopathological data of rare ANXA11 variants

From: Annexin A11 aggregation in FTLD–TDP type C and related neurodegenerative disease proteinopathies

Case

ANXA11 variant

Pathogenicity

GnomAD

Annexin A11 inclusions

Clinical diagnosis

Onset (y)

Age (y)

Sex

Neuropathological diagnosis

1

c.223C > T, p.P75S

Uncertain significance

Absent

Yes

PSP/FTD-NOS

65–70

70–75

M

FTLD-U

2

c.112G > A, p.G38R

Pathogenic

4.60E-05

Yes

ALS (Definite)

60–65

60–65

M

ALS

3

c.595G > A, p.G199S

Uncertain significance

8.55E-05

Yes

bvFTD

60–65

80–85

M

FTLD–TDP

4

c. 1423C > T, p.R475W

Uncertain significance

7.90E-05

No

PSP

60–65

60–65

F

PSP

5

c.1010 T > A, p.L337H

Uncertain significance

2.30E-04

No

PSP

60–65

70–75

M

PSP

  1. AD Alzheimer’s disease, ALS Amyotrophic Lateral Sclerosis, bvFTD behavioral variant Frontotemporal degeneration, FTD-NOS Frontotemporal degeneration not otherwise specified, FTLD-U Frontotemporal Lobar Degeneration with ubiquitin positive inclusions, PSP progressive supranuclear palsy. Age ranges are provided to help maintain anonymity